Bacillus
DH did a great smokies stool sample test. It showed he had low good bacteria and a "dangerous" bacteria called Bacillus Species. The only thing I can think of is that he has been taking PD for a while. There is something in PD called Bacillus subtilis. I called Great Smokies and they said they don't *think* it is the same thing. I did a google on Bacillus Species and Bacillus Subtilis comes up. The info it gives in unclear (to me).I have heard that there is something iffy about PD. I have also been taking it and recently have had painful intestinal cramps and need to be close by the bathroom. Happens every few days.
Any thoughts? The doctor wants Tom to take antibiotics for this. Aside from that...I sure don't want to keep having him take PD if it is indeed the problem, nor do I want to take it. I just wish I understood this more.
I had a similar problem. My Bifido bacteria was way down and had i high level of a bad bacteria called Hafnia Alvery (sp?) my Dr. had not heard of this bacteria and Iam going to do another test next week. I had also been taking PD
Probiotics are not the safe option most think , more is certainty not better , the amount you take is crucially important, too much and you compromise your immune system , and to boot the effect is transitory as the IS powers up against the introduction of alien bacteria ..I�ve just uploaded some info in the treatment section of my web site [below]
http://www.yeast-candida-infections-uk.co.uk/Pathogenic potential of lactobacilli.
Harty DW, Oakey HJ, Patrikakis M, Hume EB, Knox KW.
Institute of Dental Research, Surry Hills, N.S.W., Australia.
Lactobacilli are often considered to be commensal or beneficial participants in human microbial ecology and considerable research is being carried out into the effects of the use of lactobacilli as additives in both human and animal diets. However, lactobacilli also cause some human diseases (e.g. dental caries, rheumatic vascular disease, septicaemia and infective endocarditis (IE)), and have recently been identified as potential emerging pathogens in elderly and immunocompromised patients, particularly those receiving broad spectrum antibiotic therapy. The identification of potential pathogenic traits amongst lactobacilli will therefore facilitate the use of the organisms for probiotic purposes. The ability to aggregate human platelets is considered to be a possible pathogenic trait in the progression of IE. A comparison of bacterial cell surface properties amongst L. rhamnosus strains showed that platelets were aggregated by 5/5 IE strains and 8/16 laboratory strains. For the L. paracasei subsp. paracasei strains the respective numbers were 2/5 and 2/9. However two strains, morphological mutants of a non-aggregating strain, which had been re-isolated after passaging through rats were found to aggregate platelets. No loss of aggregating function occurred on extensive subculturing of IE strains. Aggregation also occurred with 11/14 strains for five other species, namely, Lactobacillus acidophilus, Lactobacillus fermentum, Lactobacillus oris, Lactobacillus plantarum and Lactobacillus salvivarius, with each species being represented indicating that the property is not uncommon in the genus. A comparison of IE and oral isolates of L. rhamnosus and L. paracasei subsp. paracasei and seven other Lactobacillus species, has shown that the binding of both fibronectin and fibrinogen by lactobacilli is greatly increased, up to 50 fold, when the pH is reduced from 7.0 to 5.0. Re-exposing the lactobacilli to a neutral pH environment releases most of the bound proteins, but the amount still remaining bound to the cell is several times more than is bound at neutral pH. Lactobacilli will also bind to the proteins that make up the extracellular matrix of endothelial cells. Lactobacilli bound significantly better to collagen types I and V than to types III and IV (p < 0.01). Further, strains isolated from IE cases, particularly L. rhamnosus strains, bound significantly better to types I and V than did �normal� strains (p < 0.02). Type V collagen has been demonstrated at the sites of endothelial damage. Thus the binding of lactobacilli, particularly L. rhamnosus to these collagen types may be of importance in the early stages of colonization of the damaged heart valve.(ABSTRACT TRUNCATED AT 400 WORDS)
PMID: 7703012 [PubMed - indexed for MEDLINE]
